With the fast development of our living standard,now most of us have
obesity.To well know it is really a necessity.Below is our obesity
quantum
resonance magnetic analyzer test report.Pls have a look at it.
| Abnormal lipid metabolism coefficient: |
1.992-3.713(-) |
1.113-1.992(+) |
| |
0.782-1.113(++) |
<0.782(+++) |
| |
| Brown adipose tissue abnormalities coefficient: |
2.791-4.202(-) |
2.202-2.791(+) |
| |
1.691-2.020(++) |
<1.691(+++) |
| |
| Hyperinsulinemia coefficient: |
0.097-0.215(-) |
0.215-0.426(+) |
| |
0.426-0.519(++) |
>0.519(+++) |
| |
| Nucleus of the hypothalamus abnormal
coefficient: |
0.332-0.626(-) |
0.626-0.832(+) |
| |
0.832-0.958(++) |
>0.926(+++) |
| |
| Triglyceride content of abnormal coefficient: |
1.341-1.991(-) |
1.991-3.568(+) |
| |
3.568-5.621(++) |
>5.621(+++) |
| |
| Abnormal lipid metabolism
coefficient: |
| Abnormal lipid metabolism is congenital or acquired
factors to bring out the abnormal lipid substances and their metabolites
produced for blood and other tissues and organs. Lipid metabolism to regulation
by the genetic, neural, body fluids, hormones, enzymes, and liver tissues and
organs can cause when these factors have abnormal lipid metabolism disorders and
organ pathophysiological changes. Specific symptoms, including:
hyperlipoproteinemia, lipid storage disease, obesity, fatty liver and so
on. |
| Brown adipose tissue abnormalities
coefficient: |
| Brown adipose tissue a thermogenic organ function,
when the body ingestion or cold stimulation, the brown fat cells, fat burning,
and to determine the level of the body's energy metabolism. Both cases were
known that the feeding induced by heat and cold induced heat production. Brown
adipose tissue thermogenesis organizations directly involved in the total
regulation of body heat, excess body heat is distributed to the in vitro energy
metabolism tends to balance. Brown adipose tissue thermogenesis of the body's
nutritional balance, and prevents the body from
obesity. |
| Hyperinsulinemia coefficient: |
| Obesity often coexist with hyperinsulinemia, but is
generally believed that the Department of hyperinsulinemia caused by obesity.
Hyperinsulinemic obese, insulin release is about three times the normal. Insulin
promote fat accumulation of a significant and it was suggested that insulin can
be used as an indicator of the overall fat content and obesity in a certain
sense can be used as monitoring factor. Plasma insulin concentration, and the
overall fat content was significantly positively
correlated. |
| Nucleus of the hypothalamus abnormal
coefficient: |
| Known human hypothalamus many animals there are two
pairs of feeding behavior nucleus. Abdominal contralateral nucleus (VMH), also
known as full central; another hunger for the ventrolateral nucleus (LHA), also
known as the hub. Full central excited satiety and antifeedant destroyed
appetite; central nervous system stimulation when hungry appetite, failure,
anorexia, poor feeding. Between regulation, mutual restraint, in a state of
dynamic equilibrium under physiological conditions, appetite regulation and
maintain normal body weight within normal range.The moment hypothalamic lesions
occur, whether the inflammatory sequelae (such as meningitis, encephalitis),
trauma, tumors and other pathological changes, such as the ventral medial
nuclear destruction, the ventrolateral nuclear function of relative
hyperthyroidism and bulimia assiduous , causing obesity. Conversely, when the
nuclear destruction of the ventrolateral, ventromedial nuclear function of the
relative hyperactivity and loss of appetite, caused weight
loss. |
| Triglyceride content of abnormal
coefficient: |
| Day consumption of caloric over consumption of
energy required divided by the liver and muscle glycogen in the form of storage,
almost completely converted to fat and store in the library of body fat, mainly
triglycerides, due to limited glycogen reserves. Therefore, fat as the major
storage form of body heat. Such as recurrent excessive intake of neutral fat and
carbohydrates, fat synthesis accelerated as the external causes of
obesity. |
| |
| The test results for reference only and
not as a diagnostic conclusion. |
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